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One or more keywords matched the following items that are connected to TSOYI, KONSTANTIN
Item TypeName
Concept Glycogen Synthase Kinase 3
Concept Nitric Oxide Synthase Type II
Concept Glycogen Synthase Kinase 3 beta
Academic Article Heme-oxygenase-1 induction and carbon monoxide-releasing molecule inhibit lipopolysaccharide (LPS)-induced high-mobility group box 1 release in vitro and improve survival of mice in LPS- and cecal ligation and puncture-induced sepsis model in vivo.
Academic Article PTEN differentially regulates expressions of ICAM-1 and VCAM-1 through PI3K/Akt/GSK-3?/GATA-6 signaling pathways in TNF-a-activated human endothelial cells.
Academic Article (+)-Nootkatone and (+)-valencene from rhizomes of Cyperus rotundus increase survival rates in septic mice due to heme oxygenase-1 induction.
Academic Article Activation of PPAR-gamma by carbon monoxide from CORM-2 leads to the inhibition of iNOS but not COX-2 expression in LPS-stimulated macrophages.
Academic Article Regulation of lipopolysaccharide-induced inducible nitric-oxide synthase expression through the nuclear factor-kappaB pathway and interferon-beta/tyrosine kinase 2/Janus tyrosine kinase 2-signal transducer and activator of transcription-1 signaling cascades by 2-naphthylethyl-6,7-dihydroxy-1,2,3,4-tetrahydroisoquinoline (THI 53), a new synthetic isoquinoline alkaloid.
Academic Article The heme oxygenase-1 inducer THI-56 negatively regulates iNOS expression and HMGB1 release in LPS-activated RAW 264.7 cells and CLP-induced septic mice.
Academic Article HO-1 and JAK-2/STAT-1 signals are involved in preferential inhibition of iNOS over COX-2 gene expression by newly synthesized tetrahydroisoquinoline alkaloid, CKD712, in cells activated with lipopolysacchride.
Academic Article Stimulation of alpha7 nicotinic acetylcholine receptor by nicotine attenuates inflammatory response in macrophages and improves survival in experimental model of sepsis through heme oxygenase-1 induction.
Academic Article Carbon monoxide from CORM-2 reduces HMGB1 release through regulation of IFN-?/JAK2/STAT-1/INOS/NO signaling but not COX-2 in TLR-activated macrophages.
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  • Glycogen Synthase